Cattle pasteurellosis: symptoms and treatment. Treatment of animal pasteurellosis

The infectious nature of the disease was established by Riwalt (1877) and Bollinger in 1878. L. Pasteur isolated a pure culture of the pathogen and made the first attempt to prepare a killed vaccine. In his honor, in 1910, this microbe was named pasteurella, and the disease it caused was named pasteurellosis.

Pathogen: Pasteurella multocida and Pasteurella haemolytica - a small, G~, non-motile and non-spore-forming bacterium, located in isolation, in pairs and less often in the form of chains. Pasteurella grows well on regular nutrient media. When reseeding freshly isolated cultures, it is necessary to use media with the addition of blood serum or media obtained by enzymatic hydrolysis. The growth of bacteria in the broth causes uniform turbidity of the medium; three forms of colonies are formed on MPA: smooth (S), rough (R) and mucoid (M). Stability is insignificant. Stores in corpses for 4 months, straight sun rays kill instantly, at 70-90°C they die in 5-10 minutes.

Epizootology. Course and symptoms. All types of domestic and wild animals are susceptible, including birds and humans.

Carnivores and horses are resistant.

Incubation period: from several hours to several days.

Source of pathogen: sick and recovered animals.

Routes of transmission: aerogenic, often alimentary.

Course and symptoms. Hyperacute course - death without symptoms.

In acute cases (edematous, thoracic, intestinal forms) - depression, fever up to 42°C, lack of appetite, mucopurulent discharge from the nose, conjunctivitis, cough, hemorrhagic enteritis, swelling in the intermaxillary space, death on days 2-5; in the edematous form: damage to the tongue, chest, croup, limbs, death on 1-2 days. In young animals - intestinal damage; in pigs - redness of the skin on the lower wall of the abdomen, symptoms of pharyngitis, fever, cardiac dysfunction, asphyxia, sometimes emaciation, weakness, cough, eczema. In animals, the pathogen is found in urine, blood, and feces.

Pathological and anatomical changes in pasteurellosis. In hyperacute and acute cases, hemorrhagic diathesis is found in dead animals, on the mucous membranes and serous membranes- multiple hemorrhages and inflammatory hyperemia, the liver and kidneys are degenerated, the spleen is slightly swollen, the lymph nodes are enlarged and dark red in color. In the subcutaneous tissue there are serous-fibrinous infiltrates. The lungs are swollen, with changes characteristic initial stage lobar pneumonia. In the intestinal form - fibrinous-hemorrhagic inflammation of the stomach and intestines.

In subacute and chronic course the corpses are emaciated and anemic. On the serous membranes of the thoracic and abdominal cavities There may be dense fibrillar overlays. The peribronchial lymph nodes are enlarged, hyperemic, with many hemorrhages. In the lungs, stages of red and gray hepatization are found, in some areas there are foci of necrosis; in case of complications - purulent-fibrinous foci. The spleen is slightly enlarged, and there are foci of necrosis in the liver and kidneys. Pathogenic changes in chickens are almost the same as in mammals, and mainly depend on the course of the disease.

Diagnosis of pasteurellosis. The spleen, liver, kidneys, affected parts of the lungs with lymph nodes and tubular bone. The carcasses of small animals are sent whole. For research, material is taken from parenchymal organs, from affected lungs, and lymph nodes from edematous tissues. Imprint smears are made and stained according to Gram or Romanovsky-Giemsa in order to detect typical, bipolarly stained ovoid rods. Microscopy of fresh blood has important diagnostic value. Bacteriological examination and bioassay are carried out.

Differential diagnosis. Distinguish from anthrax, emphysematous carbuncle, piroplasmosis, classical plague pigs, faces.

Prevention and treatment. In acute cases, hyperimmune serum, antibiotics, and sulfonamide drugs are used simultaneously. It is prohibited to treat sick birds.

Prevention: use of vaccines.

Emulsified vaccine against large pasteurellosis cattle, buffaloes and sheep.

Emulsified vaccine against porcine pasteurellosis.

Concentrated polyvalent formol alum vaccine against paratyphoid, pasteurellosis, diplococcal septicemia of pigs.

Precipitated formol vaccine against pasteurellosis of pigs and sheep.

Extract-formol vaccine against pasteurellosis in rabbits.

Emulsified vaccine against mink pasteurellosis.

Emulsified vaccine against nutria pasteurellosis.

Formol vaccine against pasteurellosis in cattle and buffaloes, semi-liquid aluminum hydroxide.

Quarantine is lifted 14 days after the complete recovery of animals and the last case of pasteurellosis.

Veterinary and sanitary examination. Carcasses and slaughter products from animals sick and suspected of disease are prohibited from being released in raw form. Subject to availability degenerative processes in the muscles, the carcass with internal organs is disposed of.

At degenerative changes internal organs and carcasses - scrap.

Animal skins and hair are dried in an isolated place and transported in tightly closed containers, but not earlier than 2 weeks after they are removed.

The premises are disinfected with 2% sodium hydroxide (80-90°C), then thoroughly washed hot water and again irrigated with a 4% hot solution of caustic soda.

Pasteurellosis- it's spicy infectious disease, characterized lobar inflammation lungs, extensive serous inflammatory edema subcutaneous tissue and hemorrhagic diathesis.

Pathogen information. The causative agent of the disease is Pasteurella multoida (serotypes B, D). The short ovoid rod (0.3-1.5 µm long and 0.15-0.25 µm wide) is gram-negative, non-motile, grows well on regular nutrient media.

P. multoida is antigenically heterogeneous. Based on the results of the seroprotection reaction, 4 immunological types are distinguished - I, II, III and IV (Robert, 1947), which allows 4 serological groups of Pasteurella to be distinguished in RIGA based on the capsular antigen - A, B, D and E (Carter, 1961). The serotyping of Pasteurella has not been fully resolved.

Pasteurella resistance is low, natural conditions they die relatively quickly in manure, blood, cold water pasteurellas remain viable for 2 - 3 weeks, in corpses - up to 4 months, in frozen bird carcasses - for a year. Direct sunlight kills pasteurellas in a few minutes, at a temperature of 70 - 90 ° C they die in 5-10 minutes All well-known Disinfectants in normal concentrations are destructive. They act within a few minutes.

Under natural conditions, pasteurella often penetrates the body of animals through aerogenic and nutritional routes. There may be an endogenous infection. Pasteurella penetrates the blood and lymph and causes septicemia and lobar pneumonia.

There is a hyperacute and acute course.

Epizootological data. All species of domestic and wild mammals, animals and birds are susceptible to pasteurellosis. Humans also suffer from pasteurellosis. Among chickens and rabbits, the disease usually manifests itself as an epizootic. Epizootic outbreaks of the disease are also common in other animal species, but they are observed relatively rarely. Carnivores and horses have some resistance to pasteurellosis.

The source of the pathogen is sick and recovered animals - carriers of Pasteurella. The duration of carriage can be more than a year.

Clinical signs and course. The incubation period ranges from several hours to 2 - 3 days. In all animals, pasteurellosis can occur hyperacutely, acutely, subacutely and chronically.

In cattle and buffaloes, the hyperacute course of pasteurellosis is manifested by a sudden increase in temperature to 41-42 ° C and general septic phenomena. The death of the animal occurs within a few hours with symptoms of rapidly increasing heart failure, pulmonary edema and sometimes bloody diarrhea. The animal may die even before any clinical signs.

The acute course of pasteurellosis is most characterized by general depression of the animal, manifested by lethargy, anorexia and hyperthermia, reaching 40 °C and above. The nasal planum is cold and dry. Chewing gum and lactation stop, at the beginning of the disease, peristalsis and defecation slow down, then the stool becomes watery, sometimes mixed with fibrinous flakes and blood. Bloody nasal discharge often occurs, acute conjunctivitis and bloody urine. Animals develop a pronounced picture of septicemia, heart failure and die within 1-2 days.

With more long term illnesses, in addition common features fevers may develop local lesions; according to them clinical manifestation There are edematous, thoracic and intestinal forms of pasteurellosis. In the edematous form of the disease, rapidly increasing, painful, hot and non-creating swelling of the subcutaneous tissue appears in the area lower jaw, neck, abdomen and limbs. With swelling of the tongue and neck, breathing is wheezing and labored, viscous saliva is released; visible mucous membranes are cyanotic with multiple hemorrhages. In some animals, the disease is accompanied by agitation (pasteurella meningoencephalitis of calves).

The chest form is characterized by symptoms of lobar (fibrinous) pneumonia: depression, anorexia, ruminal atony, rapid and difficult breathing, dry painful cough and serous foamy nasal discharge. Towards the end of the illness, bloody diarrhea often appears. Most animals die on the 5th -8th day.

In the intestinal form, the main symptom is heavy defeat intestinal tract, signs of pneumonia are less pronounced. Appetite remains, but animals develop progressive anemia and general depression.

In case of chronic course in animals functional disorders breathing and digestion are less pronounced than in the intestinal form, but diarrhea gradually leads to exhaustion and cachexia.

In sheep, the acute course of pasteurellosis with its inherent general clinical signs of septicemia is observed relatively rarely. Feverish state and severe depression, as a rule, are accompanied by the development of edema of the subcutaneous tissue of the anterior part of the body and fibrinous pleuropneumonia. Animals usually die on the 2nd -5th day. The subacute and chronic course of the disease is characterized by symptoms of prolonged fibrinous pleuropneumonia, keratitis, mucopurulent rhinitis, arthritis and progressive emaciation. Pasteurellosis, caused by P. haemolytica, often manifests itself as pneumonia and less often as mastitis.

In pigs, the hyperacute and acute course of pasteurellosis is characterized by fever with an increase in temperature to 41 ° C and above, pharyngitis, strained breathing, heart failure and often swelling in the intermaxillary region and neck. Animals die due to asphyxia within 1-2 days. With a more protracted course, fibrinous pleuropneumonia develops, shortness of breath, cough and mucopurulent rhinitis appear. The disease usually ends in death on the 5th -8th day. The chronic course of pasteurellosis is manifested by symptoms of pneumonia, weakness, progressive emaciation, sometimes swelling of the joints and scabby eczema.

In birds, a hyperacute course of pasteurellosis is usually noted at the beginning of an epizootic. Birds suddenly fall and, after flapping their wings several times, die without any symptoms of illness. In the vast majority of cases, the disease is acute. Birds become lethargic, sit with drooping wings, the plumage is ruffled, the head is often tucked under the wing or thrown back. Body temperature rises to 44 °C and higher, anorexia and thirst develop. Foamy mucus is secreted from the nasal openings and beak. Then profuse diarrhea appears, sometimes bloody. The comb and beard become cyanotic in color. Breathing is intense, with moist wheezing. Birds die due to convulsions or drowsiness. In the subacute and chronic course, anemia, exhaustion, and inflammation of the joints gradually develop, followed by abscess formation. In some -gits, the earrings swell and become dense (Fig. 14 and VII); Then abscesses and necrosis appear. If the earrings are affected, the previous state of health is not affected (beard disease). Chronic tasterellosis sometimes manifests itself only by signs of rhinitis, sinusitis and the accumulation of viscous exudate around the nasal openings and on the conjunctiva.

In rabbits, the acute course of pasteurellosis is manifested by hyperthermia, depression, anorexia and symptoms of damage to the upper respiratory tract(runny nose, sneezing). Sometimes diarrhea develops. Animals weaken and die after 1-2 days. In permanently disadvantaged farms, pasteurellosis occurs chronically, with signs of rhinitis and conjunctivitis. Diarrhea, fibrinous purulent pneumonia and subcutaneous abscesses are often noted.

In fur-bearing animals (sable, fox, mink, beaver), during the acute course of the disease, severe depression and anorexia are observed. slow and unsteady gait, temperature rise to 42 °C and above. As a rule, symptoms of hemorrhagic gastroenteritis develop, especially in silver foxes. Minks develop swelling of the subcutaneous tissue in the head area, paresis and paralysis hind limbs. The duration of the disease is from 12 hours to 2 - 3 days.

Pathological changes. The hyperacute course is characterized by multiple pinpoint hemorrhages in the mucous membranes of the upper respiratory tract, gastrointestinal tract and serous membranes (pericardium, epicardium, pleura). Lymph nodes, especially at the gates of infection, are enlarged, reddened, and juicy on the cut (serous inflammation). The liver, kidneys and heart are in a state of granular dystrophy. The spleen is macroscopically unchanged (sepsis without a septic spleen).

The acute course is divided into edematous, thoracic and intestinal forms.

The edematous form is characterized by serous inflammatory edema in the subcutaneous and intermuscular tissue in the head, neck, and sometimes in the area of the external genitalia and extremities. Pinpoint hemorrhages are constantly detected in the mucous membranes of the upper respiratory tract, serous tissues (costal and pulmonary pleura) and in parenchymal organs (kidneys). In a state of serous inflammation lymph nodes head, neck and chest cavity. The liver, kidneys and heart are in a state of granular dystrophy. Catarrhal or hemorrhagic inflammation of the abomasum, stomach and small intestine.

Chest shape. With this form of pasteurellosis, the main changes are found in the organs of the chest cavity. Lungs in a state of lobar pneumonia. The pericardium, epicardium, and pleura are covered with fibrin films (fibrinous serositis). Regional bronchial and mediastinal lymph nodes are enlarged, elastic, reddened, and juicy on section (serous lymphadenitis). The liver, kidneys and heart are in a state of granular dystrophy. Necrosis may be detected in the liver. Hemorrhagic diathesis is pronounced.

Intestinal form characterized by catarrhal inflammation of the mucous membrane of the stomach and small intestine. In the mucous membranes there are pinpoint hemorrhages. In other organs and tissues, the changes are weakly expressed.

Pathological diagnosis

Edema form

1. Serous inflammatory edema of the subcutaneous and intermuscular tissue of the head, neck and chest.

2. Hemorrhagic diathesis.

3. Serous inflammation submandibular, retropharyngeal and prescapular lymph nodes.

4. Granular dystrophy liver, kidneys, myocardium.

5. Unchanged spleen.

6. Acute catarrhal-hemorrhagic gastroenteritis.

Chest shape

1. Lobarnaya lobar pneumonia.

2. Serous-fibrinous pleurisy and pericarditis.

3. Hemorrhagic diathesis.

4. Serous inflammation of the bronchial and mediastinal lymph nodes.

5. Granular dystrophy of the liver, kidneys and myocardium, focal necrosis in the liver.

6. Unchanged spleen.

7. Acute catarrhal gastroenteritis.

Diagnosis: take into account the epidemiological situation, clinical symptoms, results of autopsy and bacteriological examination.

It is necessary to differentiate in pigs - from plague complicated by pasteurellosis (non-suppurative lymphocytic encephalitis, splenic infarctions, causative agent - a virus), in cattle - from anthrax (sepsis, septic spleen, causative agent - anthrax bacillus), contagious peripneumonia (lobar pneumonia with sequestration , causative agent - mycoplasma).

Pasteurellosis - infectious disease, affecting the intestines, lungs and vital important systems body. Pasteurellosis affects birds, farm animals, wild herbivores, dogs and cats (they rarely get sick).

Pasteurellosis usually occurs from March to November, but can appear at any time of the year. There are three forms of pasteurellosis: cutaneous (focal infection of soft tissues), pulmonary and septic.

In all animals, pasteurellosis can occur hyperacutely, acutely, subacutely and chronically.

Pasteurellosis - dangerous disease, and can lead to serious complications. Starting treatment for the disease in the early stages can save the animal's life. If your pet is lying down and does not respond to the environment around him, and his body temperature is elevated, contact your veterinarian immediately. Your pet needs help.

Main signs of pasteurellosis

Symptoms of pasteurellosis depend on the degree of pathogenicity and the place where the pathogen enters the body. In the early stages, the animal lies down, does not respond to calls by name and changes around, and refuses food. In some cases, the disease begins with a rise in temperature to 41 degrees and severe diarrhea, often mixed with blood.

Breathing is heavy, with coughing and wheezing, audible at a distance from the animal. Swelling of the muscles and subcutaneous tissue often appears in the head, neck, abdomen and genital areas.

Causes and course

The disease is caused by microbes of the genus Pasteurella. The pathogen persists in water and feces until three weeks. In frozen products, mainly meat, they will remain more than a year. Microbes are resistant to acids and alkalis and die within a few minutes when boiled.

The pathogen spreads through the feces of sick animals and carcasses. Domestic rodents (mice, rats) do not suffer from pasteurellosis, but they transmit pathogens.

Infection occurs by drinking contaminated water or food, eating carcasses, or sniffing sick animals. The period from infection to the development of signs of the disease is called incubation. With pasteurellosis, it lasts up to three days.

Once in the body, microbes migrate into the blood and lymph. They multiply and cause sepsis. The body responds with an immediate increase in temperature. Cells are sent to the place where microbes multiply immune system, macrophages. The production of antibodies is enhanced. Some microbes die, releasing several groups of toxins. Some attract fluid and damage the membranes of surrounding cells. Swelling occurs and active enzymes enter the blood. Another group of toxins destroys the blood. IN different areas vessels may contain red blood cells, plasma and blood clots stuck together. Oxygen delivery throughout the body deteriorates. At the same time, toxins damage blood vessels. Part: Hemorrhages occur in internal organs, and heart failure develops.

Once the pathogen enters the lungs with air or blood, the pathogen quickly multiplies. Lung cells are damaged and pneumonia begins. Toxins produced by microbes are quickly absorbed into the blood. In the intestines, microbes multiply on the contents and damage the mucous membrane. Digestion is disrupted. The body strives to get rid of germs and speeds up bowel movements.

Within a few days, death occurs from dehydration, exhaustion and sepsis. Even with assistance, more than 50% of animals infected with pasteurellosis die.

Treatment of pasteurellosis

Treatment of pasteurellosis in domestic animals is carried out in the clinic. Manipulations are aimed at:

destruction of the pathogen;
maintaining the functioning of vital organs and systems;
reduction of action and elimination of toxins.

The destruction of the pathogen is achieved by using a special serum containing antibodies to the pathogen. Antibiotics are widely used. Tetracycline, inolir and similar drugs are used. In some cases, sulfonamide drugs are used: biseptol, metronidazole, trichopolum. Maintaining the functioning of vital organs and systems is achieved in several ways. The most important thing is to improve the delivery of oxygen throughout the body. For this purpose, blood transfusions and inhalation of mixtures containing oxygen are used. Adenosine triphosphate (ATP)-based medications may be used to improve the use of available oxygen in the body. B vitamins, cocarboxylase, and ascorbic acid are widely used.

Trimetazidine, sulfocamphocaine, mildronate, etc. can be used to maintain heart function. The choice of medication depends on the condition of the animal. Replenishment of fluid lost with diarrhea is achieved by using saline (ringer, quartasol, etc.) solutions. To maintain the vital functions of the body and extraintestinal nutrition, a 5% glucose solution, hydrolysine, lipofundin, etc. are used.

Reducing the effect and removing toxins is achieved by supporting the functioning of the liver, kidneys and the use of drugs that bind poisons. Liver function is supported by early stages enzymatic (essentiale), in later years - herbal (karsil, silybor) preparations. Canephron, lespenefril, etc. are used to maintain kidney function. Among drugs that bind poisons, glutargin, sodium thiosulfate and analogues are used.

The pathogen in an infected animal is present in feces, urine, under the claws (in cats), sputum of the nasopharynx, and is released when coughing, snorting, or with blood during bleeding.

Features of pasteurellosis

Domestic rodents (mice, rats, guinea pigs) in most cases suffer from pasteurellosis for several years without symptoms.

In birds, one form of the disease is sudden death without signs of illness.

Dogs, cats and primates (including humans) often become infected with pasteurellosis through fights and bites, even from healthy animals. In such cases, all signs of the disease do not appear.

Due to the peculiarities of urine formation in cats, the administration of sulfonamides to them in some cases accelerates the development of urolithiasis.

Cattle pasteurellosis is one of the few diseases that farmers have to deal with. This disease develops quite quickly and without taking appropriate measures, it can be fatal. Typically, one week passes from the moment of infection to the death of the animal.

Description of the disease

The causative agent of this infection is Pasteurella multocida. The disease occurs in all countries of the world. In Russia, frequent outbreaks of infection are observed in the central regions. These bacteria are found on the mucous membrane of the respiratory tract of animals. As a result of decreased immunity, the disease begins to develop.

In addition, infection can occur from contact with wild animals. Even ordinary cats are considered carriers of pasteurellosis. The bacterium itself that causes this dangerous disease is weak and unstable. For example, it can die even from direct sunlight.

Epizootological data

Almost all types of domestic animals are susceptible to this disease. Cattle, birds and rabbits are most susceptible to the bacterium. High resistance is observed in horses.

The virus is mainly transmitted through direct contact. However, it is possible that the disease can be transmitted through water, food, soil and even humans.

Infected animals excrete bacteria in their feces, blood, and milk. Infection through mucous membranes is especially common. For example, through coughing or snorting. The most common outbreaks of this disease in cattle occur from July to November.

Symptoms of the disease

Cattle pasteurellosis can take several forms: acute, subacute, hyperacute and chronic. Each form of the disease has its own symptoms. The incubation period varies from several hours to three days.

Acute form

Noted depressed state And elevated temperature. In the initial stage there may be constipation, but then the stool becomes gelatinous, often mixed with blood. Maybe nosebleed. Without a vaccine, animals die within two days from heart failure.

Subacute form

Characterized by cough and fever. Cows stop producing milk. Pus begins to come out of the nose. The animal experiences constant thirst. Pronounced swelling appears in the neck and head area. Without appropriate treatment, death occurs in about two weeks.

Ultra-acute form

Manifests sharp increase fever, often accompanied by bloody diarrhea. Pulmonary edema and heart failure are noted. Death occurs within a maximum of 12 hours.

Chronic form

Manifests itself mainly persistent diarrhea, which leads the animal to exhaustion. Without the appropriate vaccine, the disease can last about three months.

Diagnosis of pasteurellosis in cattle

The diagnosis is established on the basis of epidemiological data, clinical and laboratory studies.

For staging correct diagnosis Blood and mucus smears are taken from animals. The disease is considered established when Pasteurella multocida is isolated from the blood and lungs of the animal.

Method differential diagnosis You can exclude diseases such as:

anthrax;
malignant edema;
emphysematous carbuncle.

Treatment of pasteurellosis in cattle

Infected animals are transferred to warm and dry premises and provided with adequate and balanced diet. Antibiotics are mainly used for treatment, such as:

terramycin;
biomycin;
tetracycline;
streptomycin;
chloramphenicol.

Important! It is important to administer anti-pasteurella serum only at the first symptoms of the hyperacute stage of the disease!

Remember that only a qualified veterinarian can make a diagnosis and prescribe a course of treatment. Therefore, you should not try to treat infected animals yourself. As mentioned above, the hyperacute form of the disease can lead to the death of the animal within 12 hours! Only the intervention of an experienced veterinarian can save cattle from death and prevent infection of healthy animals.

Prevention measures

The best way to prevent pasteurellosis is timely vaccination. The vaccine helps to acquire immunity to the disease for a period of six months to a year.

Try to avoid contact with other domestic and wild animals. Just one minor scratch can trigger an outbreak of the disease.

All service personnel in direct contact with animals are required to have a change of clothes and shoes. In addition, strictly comply with all sanitary and hygienic standards.

Important! After the discovery of pasteurellosis, for a year the farm is stocked with only vaccinated cattle! This will prevent relapse of the disease!

A farm is considered clean after two weeks from the moment of universal vaccination or the death of the last infected animal.

Despite the deaths, pasteurellosis is not dangerous disease. The best remedy combating this bacterium is timely disinfection of premises and care when grazing cattle. In addition, do not neglect regular veterinary examinations of all livestock.

Pasteurellosis

This is an infectious disease of cattle and other domestic and wild animals, characterized in its acute form by signs of septicemia (a form of sepsis general infection), in which the blood contains pathogenic microorganisms without involvement in inflammatory process various organs and tissues) and hemorrhagic inflammation of the mucous membranes of the respiratory tract and intestines. The person is also sick.

The causative agent – pasteurella – is poorly resistant to action disinfectants, for example, when heated to 70–90 °C, it dies within 5-10 minutes. Maximum survival in soil and water is 26 days, in manure – 72 days.

Sick and recovered animals secrete pasteurella in external environment with nasal discharge and stool. The occurrence of the disease at any time of the year is influenced by stress factors. The routes of infection are nutritional and aerogenic. Mortality ranges from 10 to 75%.

The course of the disease is hyperacute, acute, subacute and chronic. The incubation period (the time from the moment the pathogen enters the animal’s body until the first clinical signs appear) lasts from several hours to 2–3 days, sometimes more.

In hyperacute cases, animals quickly die after an increase in body temperature and the development of diarrhea, and sometimes without showing signs.

In acute cases, the body temperature of cattle rises, shortness of breath, cough, discharge from the nostrils, and sometimes diarrhea mixed with blood (more often in young animals) appear; in addition, swelling in the head, pharynx, and neck may occur. In most cases, the animals die or the disease takes a subacute or chronic course.

The chronic course is accompanied by emaciation, anemia, swelling of the joints and legs.

The diagnosis is made on the basis of clinical and epidemiological data and results bacteriological research parts of internal organs from corpses. When making a diagnosis, paratyphoid fever and anthrax should be excluded.

Patients are administered hyperimmune serum against pasteurellosis in a dose of 60–80 ml and tetracycline antibiotics, sulfonamide drugs, and symptomatic agents.

Prevention consists of administering a vaccine to healthy individuals, immunizing all animals that have had contact with sick animals, isolating sick individuals, and disinfecting premises.

From the book of Rats author Iofina Irina Olegovna

Pasteurellosis This disease is caused by bacteria and can occur in three forms: chronic, subacute and acute. Acute (septic phenomena and hemorrhagic inflammatory processes) and subacute (bronchopneumonia) forms are very transient (up to 3 days) and, as a rule,

From the book of Mice author Krasichkova Anastasia Gennadievna

Pasteurellosis The disease is caused by bacteria of the Pasteurella genus. There are acute, subacute and chronic forms of pasteurellosis. acute form hemorrhagic inflammatory processes and septic phenomena occur. When subacute form symptoms of the disease

From the book Guinea Pigs author Kulagina Kristina Alexandrovna

Pasteurellosis The main symptom of pasteurellosis, a disease caused by the activity of microbes, is a runny nose. At first, guinea pig only moistening of the hairs around the nostrils is observed, then the animal begins to sneeze and rub its nose with its front paws. Through

From the book Decorative rabbits author Neroda Margarita

Pasteurellosis Pasteurella multocida is a well-known cause of illness and death in rabbits. The most common syndrome is various diseases upper respiratory tract. Pasteurella multocida is highly contagious and can be transmitted either directly or by

From the book Diseases of Rabbits and Nutria author Dorosh Maria Vladislavovna

Pasteurellosis Pasteurellosis, or hemorrhagic septicemia, is an infectious disease of domestic and wild animals, including rabbits and nutria, characterized in its acute course by signs of septicemia (a form of sepsis (general infection), in which there are

From the book Cattle Diseases author Dorosh Maria Vladislavovna

Pasteurellosis is an infectious disease of cattle and other domestic and wild animals, characterized in its acute form by signs of septicemia (a form of sepsis of a general infection), in which pathogenic microorganisms are present in the blood without being involved in

From the book Horse Diseases author Dorosh Maria Vladislavovna

Pasteurellosis Pasteurellosis is an infectious disease of domestic and wild animals, characterized in its acute form by signs of septicemia (a form of sepsis - a general infection in which pathogenic microorganisms are present in the blood without being involved in the inflammatory process

From the book Diseases of Sheep and Goats author Dorosh Maria Vladislavovna

Pasteurellosis Pasteurellosis is an infectious disease of small ruminants and other domestic and wild animals, characterized in its acute form by signs of septicemia (a form of sepsis (general infection), in which pathogenic microorganisms are present in the blood without

From the book Diseases of Pig author Dorosh Maria Vladislavovna

Pasteurellosis Pasteurellosis, or hemorrhagic septicemia, is an infectious disease of domestic and wild animals, characterized in its acute form by signs of septicemia (a form of sepsis (general infection), in which pathogenic microorganisms are present in the blood without

From the book Hamsters author

Pasteurellosis There are three forms of this disease, caused by bacteria of the genus Pasteurella: chronic, subacute and acute. The course of the disease depends on the degree of weakening of the hamster’s body due to vitamin deficiency, violations of keeping the animal at home, complications after

From the book of Nutria author Nesterova Daria Vladimirovna

Pasteurellosis Pasteurellosis is an acute infectious disease caused by microbes (pasteurella), which short time(3–5 days) can cover a large number of animals. In addition to nutria, pasteurellosis also affects minks, silver-black foxes, sables, river

From the book Diseases poultry author Novikova Irina Nikolaevna

Pasteurellosis An infectious contagious disease occurring in acute, subacute or chronic form, the causative agent of which is the Pasteurella bacterium. external conditions bacteria survive in droppings for about 72 days, on feathers and down - from 10 to 25 days, in grain feed - up to 45

From the book Rabbits author Lapin Yuri

Pasteurellosis (hemorrhagic septicemia) The causative agent is Pasteurella. Pasteurellosis affects rabbits of all ages, and the source of infection is sick rabbits and other animals (pigs, chickens, cattle, geese, sheep, etc.), rodents and birds. Mechanical

From the book All about rabbits: breeding, keeping, care. Practical guide author Gorbunov Viktor Vladimirovich

Pasteurellosis A contagious disease that negatively affects the ears, eyes and other organs of the animal. Infection occurs through the respiratory system. The incubation period is 5–10 hours. There are acute, subacute and chronic forms of the disease. If the course of the disease is acute, then

From the book Chickens of meat breeds author Balashov Ivan Evgenievich

Pasteurellosis A dangerous disease of adult rabbits and young animals. The infection is easily transferred from a sick animal to a healthy one through care items, feeders, hands. Signs of the disease. Rabbits die very quickly (on the first – third day), so there is no possibility

From the author's book

Pasteurellosis The disease is caused by a microorganism - Pasteurella, occurs in acute, subacute and chronic forms. Sources of infection are sick and recently recovered domestic and wild birds, rodents. Infection occurs through hatching eggs,