Central chorioretinal retinal dystrophy (CRRD) is characterized by irreversible degenerative changes, occurring in the choriocapillaris layer of the vascular system of the eye, which affect the pigment layer of the retina and Bruch's membrane. Another name for this disease is senile macular degeneration, which suggests that this type of pathology usually occurs in older people and is manifested by a violation central vision. There are two forms of chorioretinal retinal dystrophy: wet ( exudative), which occurs in 9 out of 10 cases, and dry(non-exudative), occurring in 1 out of 10 cases.

Risk group

Those most at risk from the disease are primarily older people over 60 years of age. It is known that the pathology is inherited, which is why relatives of patients with chorioretinal dystrophy also need to carefully monitor the condition of their vision. Patients with atherosclerosis are also at risk.

Reasons

Chorioretinal dystrophy can be congenital or occur as a result of injury or various infections. The disease can also develop due to microcirculation disorders in the chorioretinal layer.

Often pathological process affects people over 60 years of age who have immune or endocrine system, in patients who have undergone surgery for cataracts, as well as for those who abuse tobacco smoking.

Symptoms (signs)

In the case of a non-exudative form of pathology, the quality of vision may remain unchanged for a long time, after which the manifestation of metamorphopsia is observed - a characteristic distortion of straight lines. In the future, a central scotoma may appear - a lesion that falls out of sight. The atrophic process of the choriocapillaris layer and pigment epithelium continues, which causes a significant decrease in central vision.

The exudative form of the disease is characterized by patient complaints about an image visible as if through a layer of water, curvature of straight lines, the appearance of translucent spots and flashes of light before the eyes. If left untreated, the next stage of the disease can manifest itself as a sharp decrease in vision, during which patients are practically deprived of the ability to read and write.

Diagnostic methods

It is important for the diagnosis of pathology to have characteristic features: distortion of straight lines and loss of lesions from the field of view. Distortion is assessed using the Amsler test, and campimetry is used to examine central vision. It is possible to conduct studies of contrast, color perception and the size of visual fields.

To get more detailed information Laser scanning or optical coherence tomography, vascular fluorescein angiography and electroretinography will be used.

Treatment

Therapy for chorioretinal retinal dystrophy is aimed at stabilizing the processes of tissue degeneration. You should not expect treatment to restore the quality of your vision.

To treat the disease, medications, lasers and in rare cases– surgical methods. In the non-exudative form of the disease, drugs are used to slow down the processes of degeneration, and the retina is stimulated using a helium-neon laser.

In the case of the exudative form, dehydration therapy is carried out, and laser coagulation of the retina and subretinal membranes is used, for which it is recommended to use a krypton laser. Good result give intravitreal (intraocular) injections of drugs such as: Lucentis, Avastin, Eylea.

With regard to restoration of vision quality, the prognosis is unfavorable.

Study peripheral parts- the process is not easy, often causing some difficulties. This is due to its location - in the depths of the eye, but it is there that a fairly common ophthalmic disease, called peripheral chorioretinal dystrophy. With it, the affected areas of the retina become much thinner, gaps form, which leads to very serious complication- retina.

Typically, this disease more often affects people suffering from eye pain, because longitudinal stretching of the eye can provoke severe changes in the retina, as well as other tissues of the organ of vision. It has been found that people with a higher degree of myopia are more likely to develop retinal dystrophy.

Classification of PCRD

Peripheral retinal dystrophy can be acquired or hereditary. It is detected with equal frequency in both young and elderly people. The area of ​​localization of the disease is decisive for its type. This is how it is customary to distinguish:

Chorioretinal dystrophy - involving the retina and;

Vitreoretinal dystrophy - involving the retina and.

At the same time, peripheral dystrophy subdivided into: lattice, cystic, frost-like.

Signs of the disease

The initial stage of dystrophy is asymptomatic with no patient complaints, which makes the disease especially dangerous. After all, a person pays attention to changes in vision only during the period of complications - ruptures and retinal detachment. The process of detachment is accompanied by the appearance of unexpected flashes, lightning, and fluttering “flies” before the eyes. In addition, a sign of a developed disease becomes sharp drop visual acuity and a volumetric spot that appears before the eyes. If these manifestations occur, you must immediately contact an ophthalmologist, otherwise irreversible blindness may occur.

Video of a specialist about PCRD

Diagnosis of peripheral retinal dystrophies

In the diagnosis of peripheral dystrophy, a three-mirror Goldmann lens is used, which makes it possible to examine areas invisible with a conventional ophthalmoscope, which reveals the most dangerous violations. The inspection is carried out with an expanded view. Often, for a more detailed study of the affected area, a method is used that involves retracting the retina to the middle, which makes it possible to inspect peripheral areas.

With initial modifications of the retina, treatment is not necessary, but this period requires dispensary observation behind the patient and periodically photographing the periphery using a slit lamp.

Treatment and prevention of peripheral dystrophies

Establishing a diagnosis of peripheral retinal dystrophy involves long-term follow-up for illness or laser treatment. In case laser treatment the affected areas of the retina are fenced off from the center with an artificial barrier, which prevents further detachment. This method is considered to be particularly effective, because it eliminates the need to use surgical intervention. Laser treatment, at its core, is effective prevention retinal detachment. This procedure does not cause pain, is easily tolerated, takes less than 15 minutes. It is performed on an outpatient basis and does not cause any damage to the patient’s vision. On late stages the disease requires complex surgical intervention, which advanced cases, often does not give the expected result, and it is not possible to completely restore vision.

Prevention of this disease mandatory for patients suffering from myopia; diabetes mellitus; children born in difficult birth; persons who have genetic predisposition to peripheral dystrophies.

PCRD and pregnancy

Peripheral retinal dystrophies may serve as a contraindication to childbirth naturally. To prevent the retina during the pressing period (or during everyday life) carry out PPLC - preventive peripheral or limiting laser coagulation of the retina (depending on the severity of changes in the fundus). They can be performed during pregnancy - there are no contraindications.

It is worth understanding that the ophthalmologist’s opinion is only one of the factors that the obstetrician takes to decide on the method of delivery (along with the size of the pelvis, fetus, etc.).

Where to go for treatment

Laser treatment for peripheral retinal dystrophies is available today in many large ophthalmology centers. And yet, choosing a clinic for yourself is a very important moment, because the prognosis for recovery, as well as the result of treatment, will largely depend on the quality of the examination and professional excellence attending physician.

By contacting any of the well-known eye clinics in Moscow, you are guaranteed to see excellent diagnostic doctors, and then, according to indications, you will be referred to a highly qualified specialist in laser surgery. Ophthalmological centers only work the best professionals with extensive experience behind them clinical practice. And the technical equipment of the clinic allows us to guarantee excellent results even in the most difficult cases of eye diseases.

Chorioretinal dystrophy of the retina is a disease that causes irreversible destruction of the tissue of the vascular layers eyeball, during which vision deteriorates significantly. This is why it is so important to undergo annual examinations with an ophthalmologist to prevent or detect early stages the beginning of destructive processes in the retinal tissue, which can develop with virtually no symptoms.

During the development of dystrophy, the level of central vision decreases significantly. However, the lesion does not affect the peripheral vision area, which continues to function stably. Thanks to this, even with severe forms of the disease, the patient will be able to navigate normally in a normal environment, although he will not be able to cope with activities such as driving or reading without additional devices that correct vision.

The standard examination procedure by an ophthalmologist does not allow us to examine the condition of the peripheral zone of the eyes. However, it is precisely in this area of ​​the retina that tissue is most often affected degenerative processes. Since it is not possible to detect them and begin treatment immediately due to their practically asymptomatic course, the patient may face a number of additional complications, which ultimately lead to serious visual impairment, for example, tissue rupture or detachment.

This disease most often occurs in older people with light pigmentation of the iris. The structure of their blood vessels is heavily exposed age-related changes, as a result of which the retinal tissue begins to wear out intensively. In this case, the destruction process is significantly accelerated if the patient has bad habits(alcohol, smoking).

Chorioretinal retinal dystrophy

Based on type and development (pathogenesis), the following CCRDs are distinguished:

1. Dry atrophic (non-exudative) - early form a disease in which decreased vision is accompanied by the death of the pigment epithelium. At the same time, you need to be prepared for the fact that in the next 5 years the lesion will begin to actively develop in the second eye.
2. Wet (exudative). It is quite rare. Complicated by detachments various types epithelium, hemorrhages in the area of ​​affected tissues and cicatricial deformation.

As a rule, such changes can be observed even in people whose vision is normal.

Manifestations of the disease

As a rule, central chorioretinal dystrophy of the retina is classified as a multifactorial pathology that can be provoked by:

• bad heredity;
• various stages of myopia;
• violations in vascular system eye;
• weakening of the immune system;
• intoxication by various chemicals or alcohol;
• vitamin deficiency;
• concomitant diseases, such as diabetes.

The disease can begin to develop after various eye injuries, including cataracts and surgery.

On initial stages Tissue degeneration in the retina, as a rule, occurs without noticeable symptoms. The first of them appear only in the moderate or severe stages of the disease. Thus, in patients with a dry form of dystrophy, during the deterioration of vision, a veil, floaters or white flashes may periodically appear before the eyes.

The wet form of dystrophy is expressed by severe distortion or blurriness of surrounding objects, the development of color blindness (impaired color discrimination) and erroneous perception of objects in space.

If CCRD is not detected promptly and treatment is not started, tissues continue to deteriorate and vision deteriorates sharply. The disease can be detected by examining the fundus of the eye. For this purpose, a special three-mirror Goldmann lens is most often used, which allows you to see even the most extreme areas of the retina.

Additional diagnostic methods:

• conducting optical coherence and laser scanning tomography of the retina;
• implementation of computer perimetry;
• electroretinography;
• fluorescein angiography of retinal vessels.

In the early stages of diagnosing CCRD, the doctor can use a number of special tests that allow them to study how much the patient’s color rendition and contrast of vision are impaired.

Treatment options

Most drugs that help slow or stop the development of CCRD are taken in courses in accordance with doctor’s prescriptions:

Depending on the stage at which the disease was detected, drug treatment can be supplemented with physiotherapeutic procedures.

Laser treatment is often used, which is a series of therapeutic techniques based on the effect of laser on the affected areas of the retina without affecting its healthy parts. The affected areas are irradiated to activate them metabolic processes. At the same time blood vessels are sealed, blood and fluid stop appearing under the retina, peeling it off, thereby stopping the development of the disease. The treatment procedure is painless and does not take much time.

During surgical laser intervention, the tissue in the affected area of ​​the retina is glued (sealed) with a laser beam, which makes it possible to isolate them from healthy tissue and stop further development of the disease. It is worth noting that surgical intervention for CCRD is carried out if the disease takes severe form, at which laser correction or special drugs do not give results.

All operations are divided into two categories:

• revascularization - removal of damaged vessels and maximum opening of normal ones;
• vasoreconstructive - the microvascular beds of the eyes are restored using grafts.

The patient in mandatory You will need to visit an otolaryngologist and dentist. These doctors will have to confirm the absence of chronic foci of infection and draw up appropriate conclusions, on the basis of which the ophthalmologist will give permission to perform the operation.

Possible complications

Even in severe form, chorioretinal retinal dystrophy does not cause blindness, therefore, even despite certain difficulties, a person will be able to determine the basic characteristics of objects and see their movement in space, if this occurs.

Particular attention to vision control should be paid to women who are in the second trimester of pregnancy. During this period, their blood pressure decreases significantly compared to normal, disrupting stable work circulatory system.

As a result, the flow of blood and nutrients to eye vessels in some women it is disrupted, provoking the onset of degenerative processes. Moreover, the risk of their development increases significantly if a woman was treated for any eye disease before conception.

To avoid unforeseen complications, a pregnant woman should definitely be examined by an ophthalmologist. If the risk of instant dystrophy during childbirth is high, then a caesarean section will be performed to avoid fetal loss.

In addition, if a person is at risk, he must undergo an annual fundus examination by an ophthalmologist. We should also not forget about therapeutic courses aimed at stabilizing the functioning of the vascular system.

These procedures will improve peripheral circulation, stimulation of metabolic processes in the retinal tissue, which will allow the patient to even read using special devices, such as a magnifying glass or telescopic glasses, which magnify the image transmitted to the retina.

In this case, it is imperative to carry out daily monitoring of the level blood pressure and protect your eyes from exposure ultraviolet rays. These same methods of protecting vision serve as the main preventive measures for healthy people.

Video

Central disiform dystrophy of the Kunt-Junius type, etc.) is a bilateral chronic dystrophic process with predominant damage to the choriocapillary layer of the choroid, Bruch's vitreous membrane (the border plate between the retina and the choroid) and the retinal pigment epithelium layer. Prevalence in the general population increases with age: 1.6% in those aged 51–64 years, 11% in those aged 65–74 years and 27.9% in those over 75 years of age, more common in women; is the leading cause of irreversible loss of central vision in the second half of life.

Symptoms:

In the non-exudative form, at first there are either no complaints, or metamorphopsia (curvature of straight lines) may be observed, visual acuity remains unchanged for a long time; then the central one develops (i.e., the area of ​​loss of the visual field within its boundaries) due to the choriocapillaris layer and pigment epithelium, manifested by some “shading” of objects in the center of the visual field; Central visual acuity is significantly reduced. Objectively, multiple small, round or oval, slightly protruding foci of white or yellow, sometimes bordered by a pigment rim, in places merging into yellowish-white conglomerates, separated by brown lumps of pigment. The process may stabilize. The fellow eye may develop exudative form TsHRD.
The exudative form of CCRD goes through 5 stages in its development: 1) the stage of exudative detachment of the pigment epithelium - central visual acuity remains high (0.8-1.0), perhaps transiently weak or; in some cases there are complaints of curvature, wavy straight lines, a feeling of looking “through a layer of water” (metamorphopsia), a translucent single spot or group dark spots before the eye (relative positive scotoma), flashes of light (photopsia). Retina in the area macular spot slightly raised into the vitreous body in the form of a dome with clear yellowish borders, drusen become invisible; the detachment may adhere independently; 2) stage of exudative detachment of the neuroepithelium - vision is reduced to a greater extent, other complaints are the same, however, the clarity of the boundaries of the detachment decreases, the raised retina is swollen; 3) stage of neovascularization - sharp decline visual acuity up to 0.1-0.2 or up to hundredths, loss of the ability to read and write; ophthalmoscopically, the area of ​​the retina in the area of ​​the macula acquires a dirty gray color, racemose edema of the neuroepithelium and hemorrhages under the retina or in the vitreous body appear; FA examination records the fluorescence of newly formed vessels (subretinal neovascular membrane) in the form of a “lace”; 4) stage of exudative-hemorrhagic detachment of the pigment and neuroepithelium - vision remains low, a disc-shaped lesion up to several disc diameters has formed in the macular area optic nerve white-pink or gray-brown color with pigment deposits and newly formed vessels, racemose-shaped retina, with clear boundaries and prominence into the vitreous body; 5) the scar stage is characterized by the development of fibrous tissue.
The diagnosis is established on the basis of the patient’s characteristic complaints (curvature of straight lines and other types of metamorphopsia), research visual functions(central visual acuity, campimetry, nine-point test or Amsler grid), ophthalmoscopy, fluorescein angiography of the fundus.
Sometimes it is necessary to carry out differential diagnosis with choroidal melanosarcoma.
The disease usually appears after age 60, first in one eye, and after about 4 years similar changes develop in the fellow eye. The clinical course is chronic, slowly progressive. The severity of the disease is determined by bilateral damage, central localization of the process in the fundus and a serious decrease in the quality of life due to the loss of the ability to read and write.

Causes:

The etiology is unknown, although the familial, hereditary nature of the process with an autosomal dominant type of inheritance has been established. Risk factors include ultraviolet radiation, which leads to the accumulation of toxic metabolic products (free radicals) in the retinal epithelium and the formation of colloidal drusen.
Central chorioretinal dystrophy (CCRD) can be classified as amyloidosis of the posterior segment of the eye: the dystrophic process begins with the appearance of accumulations of hard or soft colloidal substance (the so-called drusen) between Bruch's membrane and the retinal pigment epithelium in the macula and paramacular zone. The pigment epithelium in places of contact with the solid colloidal substance becomes thinner, loses pigment, and in neighboring areas it thickens and hyperplasia occurs. Bruch's membrane is of uneven thickness with calcification of its elastic and collagen fibers, and in the choriocapillary layer of the underlying choroid, thickening and hyalinization of the stroma occurs (non-exudative, atrophic or dry form of CCRD). “Soft” drusen can cause exudative detachment of the pigment epithelium, and then the retinal neuroepithelium (exudative or disciform form of CCRD). Further development of the process in this variant of pathology is accompanied by the development of a membrane of newly formed vessels under the retina and the transition of the disease to the exudative-hemorrhagic stage with the appearance of hemorrhages under the pigment epithelium, in the subretinal space or (rarely) in the vitreous body. Subsequently, resorption of hemorrhage occurs and development of fibrous scar tissue.
According to pathogenesis they distinguish following forms CCRD: non-exudative (dry, atrophic) - 10-15% of cases and exudative (disciform) - 85-90% of cases. Stages clinical course exudative CCRD is distinguished depending on the type of exfoliated retinal epithelium (pigment or neuroepithelium) and the nature of the subepithelial contents (exudate, neovascular membrane, blood, fibrous tissue) - see more in the description of symptoms.

Treatment:

For treatment the following is prescribed:

Treatment can be medication, laser and, less commonly, surgery. It is aimed at stabilizing and compensating the process, since full recovery normal vision impossible. For the atrophic non-exudative form, antiplatelet agents, angioprotectors, antioxidants and vasodilators(Cavinton) 2 courses per year - (spring and autumn), the retina is stimulated with a defocused beam of a helium-neon laser. For exudative disciform CCRD, local and general dehydration therapy, laser coagulation of the retina and subretinal neovascular membranes, preferably with a krypton laser, are prescribed. Surgical methods treatments are aimed either at improving the blood supply to the posterior segment of the eye (revascularization, vasoreconstruction in non-exudative form), or at removing subretinal neovascular membranes. If macular degeneration is combined with cataracts, removal of the cloudy lens is carried out according to a known method, but instead of a conventional artificial lens, special intraocular lenses can be implanted, which shift the image to the unaffected area of ​​the retina (spheroprismatic lenses) or provide an enlarged image on the retina (bifocal lenses).
Critical visual acuity favorable for treatment is 0.2 or higher. In general, the prognosis for vision is unfavorable.

Getting worse peripheral vision and dark adaptation. This means that they have difficulty seeing in dark rooms and have difficulty distinguishing objects located to the side of them. As complications develop, patients may experience more obvious and dangerous symptoms.

Species

Depending on the nature of the pathological changes in the retina, several types of peripheral degeneration are distinguished. Only an ophthalmologist can distinguish them after performing an ophthalmoscopy - examination of the fundus of the eye. During the examination, the doctor can see the retina, which lines the inside of the eyeball.

Lattice

Occurs in approximately 65% ​​of cases and has the most poor prognosis. According to statistics, it is lattice dystrophy that most often leads to retinal detachment and loss of vision. Fortunately, the disease has an indolent course and progresses slowly throughout life.

Lattice degeneration is characterized by the appearance of narrow white stripes on the fundus of the eye, which in appearance resemble a lattice. They are empty vessels of the retina filled with hyaline. Between the white cords, areas of thinned retina that have a pinkish or red tint are visible.

According to the type of snail trail

This type of peripheral retinal degeneration most often develops in people with high degrees of myopia. The pathology is characterized by the appearance of peculiar holey defects on the retina, which outwardly resemble a snail's footprint on the asphalt. The disease leads to ruptures of the retina with its subsequent detachment.

Frost-like

It is a hereditary disease that affects men and women. Frost-like peripheral dystrophy is characterized by the appearance of whitish deposits on the retina of the eye, which outwardly resemble snow flakes.

Cobblestone type

Refers to the least dangerous retinal PVCRD. Occurs more often in older people and people with. The disease is practically asymptomatic and has a relatively favorable course. It extremely rarely leads to ruptures or.

In cobblestone dystrophy, multiple foci of degeneration are visible in the fundus. They have white, elongated shape and uneven surface. As a rule, all lesions are located in a circle, at the very periphery of the fundus.

Retinoschisis

The disease is hereditary. It leads to delamination of the retina and the formation of huge cysts filled with fluid. Retinoschisis is asymptomatic. In some cases, it is accompanied by local loss of vision at the location of the cyst. But since the pathological foci are on the periphery, this goes unnoticed.

Small cystic

The pathology is also called Blessin-Ivanov disease. Small cystic retinal retinal retinal retina is accompanied by the formation of many small cysts on the periphery of the fundus. Usually the disease has a slow course and a favorable prognosis. However, in rare cases, cysts can rupture, causing retinal tears and detachments.

Reasons

A considerable part of peripheral chorioretinal dystrophies of the retina refers to hereditary diseases. In 30-40% of cases, the development of the disease provokes high degrees of myopia, in 8% -. Retinal damage can also result from systemic or ophthalmic diseases.

Possible causes of the development of peripheral degenerations:

• hereditary predisposition, the presence of PVCD in close relatives;
• myopia (myopia) of any degree;
• inflammatory eye diseases (endophthalmitis, iridocyclitis, etc.);
• previous injuries and surgical interventions;
• diabetes mellitus and other diseases of the endocrine system;
• intoxication and viral infections;
• atherosclerosis and diseases of the cardiovascular system;
• frequent and prolonged exposure to the sun without sunglasses;
• lack of vitamins and minerals that are necessary for normal functioning retina of the eye.

In young people, acquired retinal degenerations most often develop against the background of high myopia. In older people, pathology occurs due to disruption of normal blood circulation and metabolism in the tissues of the eyeball.

Symptoms

At first, peripheral retinal dystrophy does not manifest itself in any way. Occasionally, it may make itself felt with bright flashes or the flickering of spots before the eyes. If the patient has impaired lateral vision, he for a long time doesn't notice this. As the disease progresses, a person realizes that he needs brighter light when reading. Over time, he may develop other visual impairments.

Most frequent symptoms PVHRD:

• Narrowing of visual fields. The patient has difficulty seeing objects located on the periphery. He has to turn his head to see them. In the most severe cases, people develop what is called tunnel vision.
• The appearance of cattle. Defects in the visual field may not be noticeable or may appear as black or colored spots. In the first case, they can be identified using additional methods research – perimetry and campimetry.
• Nyctalopia. Peripheral retinal degeneration leads to damage to the rods - photosensitive elements responsible for night vision. For this reason, pathology is often accompanied night blindness (poor eyesight at dusk).
• Metamorphopsia. The symptom is manifested by distortion of the contours and sizes of visible objects.
• Blurred vision. A person may feel as if he is looking at the world through a fog or thick layer water.

When complications occur (retinal rupture or detachment), the patient experiences sparks, lightning, and bright flashes before the eyes. Subsequently, a dark curtain forms in the field of vision, preventing normal vision. These symptoms are extremely dangerous, so if they occur, you should immediately consult a doctor.

At-risk groups

Peripheral chorioretinal retinal dystrophy most often affects people with a high degree of myopia. This is due to the fact that with myopia the eyeball grows in length. Consequently, this leads to severe stretching and thinning of the retina. As a result, it becomes extremely susceptible to dystrophic processes.

The risk group also includes people over 65 years of age and patients with diabetes mellitus, atherosclerosis, hypertension. All these people have retina various reasons suffers from a lack of oxygen and nutrients. All this is a powerful impetus for the development of dystrophy.

Which doctor treats peripheral retinal dystrophy?

The treatment of retinal retinal retina is performed by a retinologist. He specializes in the diagnosis and treatment of diseases of the posterior segment of the eyeball ( vitreous, retina and choroid).

If complications develop, the patient may require the help of an ophthalmic surgeon or laser ophthalmologist. These specialists perform complex operations, allowing to cure retinal rupture and detachment. IN difficult situations their help allows you to preserve and even restore partially lost vision.

Diagnostics

Methods used to diagnose peripheral degenerations.

Treatment

To date, there are no methods that can completely cure peripheral vitreochorioretinal dystrophy of the retina. The progression of the disease can be stopped with the help of medication, physiotherapy, laser and surgical treatment. The correct approach to combating the disease allows you to preserve your vision and avoid dangerous complications.

Drugs that help slow the progression of dystrophy:

• Antiplatelet agents ( Acetylsalicylic acid, Ticlopidine, Clopidogrel). Useful for hypertension and cardiovascular diseases. They thin the blood and inhibit thrombus formation, thereby protecting the retina from hypoxia.
• Vasodilators and angioprotectors (Actovegin, Vinpocetine, Pentoxifylline). Relieves spasm of retinal vessels and protects vascular walls from damage. They improve blood circulation in the retina, preventing its destruction.
• Vitamin and mineral complexes (Okyuvite-Lutein, Blueberry-Forte). Saturate the retina nutrients, vitamins and minerals. Slow down the course of degenerative processes.
• Biological regulators (Retinolamine). Restore the permeability of retinal vessels and normalize the functioning of photoreceptors. They stimulate reparative processes, that is, they accelerate the restoration of the retina.

You can strengthen the retina and prevent its detachment using laser treatment. During the procedure, the specialist solders the retina to the choroid, which is located underneath it. This allows you to limit the foci of degeneration.

Prevention

There is currently no specific prevention of the disease. You can reduce the risk of developing dystrophy with the help of eye exercises and proper nutrition. Very useful in this regard biologically active additives, containing zinc, vitamins E, A, group B. Protecting your eyes from ultraviolet radiation with sunglasses also helps to avoid illness.

Peripheral retinal dystrophies often develop in older people and in people with high degrees of myopia. The reason for this is stretching of the retina or impaired blood circulation and metabolism in it.

To combat degenerations, medications, physiotherapy, laser and surgical techniques. Laser coagulation retina today is considered the most effective method treatment of pathology. With its help, it is possible to limit dystrophic foci and prevent the spread of the pathological process.

Useful video about peripheral retinal dystrophy